Signal termination of the chemokine receptor CCR9 is governed by an arrestin-independent phosphorylation mechanism.

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Bibliographic Details
Title: Signal termination of the chemokine receptor CCR9 is governed by an arrestin-independent phosphorylation mechanism.
Authors: Lamme TD; Faculty of Science, Division of Medicinal Chemistry, Department of Chemistry and Pharmaceutical Sciences, Amsterdam Institute for Molecular and Life Sciences (AIMMS), Vrije Universiteit Amsterdam, Amsterdam, The Netherlands., Smit MJ; Faculty of Science, Division of Medicinal Chemistry, Department of Chemistry and Pharmaceutical Sciences, Amsterdam Institute for Molecular and Life Sciences (AIMMS), Vrije Universiteit Amsterdam, Amsterdam, The Netherlands., Schafer CT; Faculty of Science, Division of Medicinal Chemistry, Department of Chemistry and Pharmaceutical Sciences, Amsterdam Institute for Molecular and Life Sciences (AIMMS), Vrije Universiteit Amsterdam, Amsterdam, The Netherlands. Electronic address: c.t.schafer@vu.nl.
Source: The Journal of biological chemistry [J Biol Chem] 2025 May; Vol. 301 (5), pp. 108462. Date of Electronic Publication: 2025 Mar 26.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Elsevier Inc. on behalf of American Society for Biochemistry and Molecular Biology Country of Publication: United States NLM ID: 2985121R Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1083-351X (Electronic) Linking ISSN: 00219258 NLM ISO Abbreviation: J Biol Chem Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:1083-351X
DOI:10.1016/j.jbc.2025.108462