The RNA-binding protein CELF1 targets ATG5 to regulate autophagy and promote drug resistance in acute myeloid leukemia.
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| Title: | The RNA-binding protein CELF1 targets ATG5 to regulate autophagy and promote drug resistance in acute myeloid leukemia. |
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| Authors: | Li X; School of Basic Medical Sciences & Forensic Medicine, Hangzhou Medical College, Hangzhou, China., Qian Q; School of Basic Medical Sciences & Forensic Medicine, Hangzhou Medical College, Hangzhou, China., Li J; School of Basic Medical Sciences & Forensic Medicine, Hangzhou Medical College, Hangzhou, China., Zhang L; School of Life Sciences, Zhejiang Chinese Medical University, Hangzhou, China., Wang L; School of Basic Medical Sciences & Forensic Medicine, Hangzhou Medical College, Hangzhou, China., Huang D; School of Basic Medical Sciences & Forensic Medicine, Hangzhou Medical College, Hangzhou, China. dshuang@hmc.edu.cn.; Zhejiang Key Laboratory of Tumor Molecular Diagnosis and Individualized Medicine, Zhejiang Provincial People's Hospital, Affiliated People's Hospital, Hangzhou Medical College, Hangzhou, Zhejiang, China. dshuang@hmc.edu.cn., Xu Q; School of Basic Medical Sciences & Forensic Medicine, Hangzhou Medical College, Hangzhou, China. xuqiuran@hmc.edu.cn.; Zhejiang Key Laboratory of Tumor Molecular Diagnosis and Individualized Medicine, Zhejiang Provincial People's Hospital, Affiliated People's Hospital, Hangzhou Medical College, Hangzhou, Zhejiang, China. xuqiuran@hmc.edu.cn., Chen W; School of Basic Medical Sciences & Forensic Medicine, Hangzhou Medical College, Hangzhou, China. chenwenhu@hmc.edu.cn. |
| Source: | Cell death & disease [Cell Death Dis] 2025 Aug 08; Vol. 16 (1), pp. 599. Date of Electronic Publication: 2025 Aug 08. |
| Publication Type: | Journal Article |
| Journal Info: | Publisher: Nature Pub. Group Country of Publication: England NLM ID: 101524092 Publication Model: Electronic Cited Medium: Internet ISSN: 2041-4889 (Electronic) NLM ISO Abbreviation: Cell Death Dis Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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| ISSN: | 2041-4889 |
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| DOI: | 10.1038/s41419-025-07926-0 |
