Genetic inhibition of IL-12β suppresses systolic overload-induced cardiac inflammation and heart failure development.

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Bibliographic Details
Title: Genetic inhibition of IL-12β suppresses systolic overload-induced cardiac inflammation and heart failure development.
Authors: Bhattarai U; Department of Physiology and Biophysics, School of Medicine, University of Mississippi Medical Center, Jackson, MS, United States., He X; Department of Physiology and Biophysics, School of Medicine, University of Mississippi Medical Center, Jackson, MS, United States., Niu Z; Department of Physiology and Biophysics, School of Medicine, University of Mississippi Medical Center, Jackson, MS, United States., Pan L; Department of Physiology and Biophysics, School of Medicine, University of Mississippi Medical Center, Jackson, MS, United States., Wang D; Department of Physiology and Biophysics, School of Medicine, University of Mississippi Medical Center, Jackson, MS, United States., Wang H; Department of Physiology and Biophysics, School of Medicine, University of Mississippi Medical Center, Jackson, MS, United States., Zeng H; Department of Pharmacology and Toxicology, School of Medicine, University of Mississippi Medical Center, Jackson, MS, United States., Chen JX; Department of Pharmacology and Toxicology, School of Medicine, University of Mississippi Medical Center, Jackson, MS, United States., Speed JS; Department of Physiology and Biophysics, School of Medicine, University of Mississippi Medical Center, Jackson, MS, United States., Clemmer JS; Department of Physiology and Biophysics, School of Medicine, University of Mississippi Medical Center, Jackson, MS, United States., Hall JE; Department of Physiology and Biophysics, School of Medicine, University of Mississippi Medical Center, Jackson, MS, United States., Chen Y; Department of Physiology and Biophysics, School of Medicine, University of Mississippi Medical Center, Jackson, MS, United States.
Source: BioRxiv : the preprint server for biology [bioRxiv] 2025 Sep 11. Date of Electronic Publication: 2025 Sep 11.
Publication Type: Journal Article; Preprint
Journal Info: Country of Publication: United States NLM ID: 101680187 Publication Model: Electronic Cited Medium: Internet ISSN: 2692-8205 (Electronic) Linking ISSN: 26928205 NLM ISO Abbreviation: bioRxiv Subsets: PubMed not MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:2692-8205
DOI:10.1101/2025.09.05.674485