MYBPC3 N-terminal missense mutations linked to hypertrophic cardiomyopathy strengthen actin binding and enhance residue mobility.

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Bibliographic Details
Title: MYBPC3 N-terminal missense mutations linked to hypertrophic cardiomyopathy strengthen actin binding and enhance residue mobility.
Authors: Kanassatega RS; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, Arizona, USA., Bunch TA; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, Arizona, USA., Wong FL; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, Arizona, USA., Espinoza-Fonseca LM; Department of Internal Medicine, University of Michigan, Ann Arbor, Michigan, USA., Colson BA; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, Arizona, USA. Electronic address: bcolson@arizona.edu.
Source: The Journal of biological chemistry [J Biol Chem] 2025 Dec; Vol. 301 (12), pp. 110855. Date of Electronic Publication: 2025 Oct 22.
Publication Type: Journal Article; Research Support, N.I.H., Extramural
Journal Info: Publisher: Elsevier Inc. on behalf of American Society for Biochemistry and Molecular Biology Country of Publication: United States NLM ID: 2985121R Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1083-351X (Electronic) Linking ISSN: 00219258 NLM ISO Abbreviation: J Biol Chem Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:1083-351X
DOI:10.1016/j.jbc.2025.110855