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MYBPC3 N-terminal missense mutations linked to hypertrophic cardiomyopathy strengthen actin binding and enhance residue mobility.

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Title: MYBPC3 N-terminal missense mutations linked to hypertrophic cardiomyopathy strengthen actin binding and enhance residue mobility.
Authors: Kanassatega RS; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, Arizona, USA., Bunch TA; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, Arizona, USA., Wong FL; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, Arizona, USA., Espinoza-Fonseca LM; Department of Internal Medicine, University of Michigan, Ann Arbor, Michigan, USA., Colson BA; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, Arizona, USA. Electronic address: bcolson@arizona.edu.
Source: The Journal of biological chemistry [J Biol Chem] 2025 Dec; Vol. 301 (12), pp. 110855. Date of Electronic Publication: 2025 Oct 22.
Publication Type: Journal Article; Research Support, N.I.H., Extramural
Journal Info: Publisher: Elsevier Inc. on behalf of American Society for Biochemistry and Molecular Biology Country of Publication: United States NLM ID: 2985121R Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1083-351X (Electronic) Linking ISSN: 00219258 NLM ISO Abbreviation: J Biol Chem Subsets: MEDLINE
Database: MEDLINE Ultimate
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PubType: Academic Journal
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  Data: MYBPC3 N-terminal missense mutations linked to hypertrophic cardiomyopathy strengthen actin binding and enhance residue mobility.
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  Data: <searchLink fieldCode="AU" term="%22Kanassatega+RS%22">Kanassatega RS</searchLink>; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, Arizona, USA.<br /><searchLink fieldCode="AU" term="%22Bunch+TA%22">Bunch TA</searchLink>; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, Arizona, USA.<br /><searchLink fieldCode="AU" term="%22Wong+FL%22">Wong FL</searchLink>; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, Arizona, USA.<br /><searchLink fieldCode="AU" term="%22Espinoza-Fonseca+LM%22">Espinoza-Fonseca LM</searchLink>; Department of Internal Medicine, University of Michigan, Ann Arbor, Michigan, USA.<br /><searchLink fieldCode="AU" term="%22Colson+BA%22">Colson BA</searchLink>; Department of Cellular and Molecular Medicine, University of Arizona, Tucson, Arizona, USA. Electronic address: bcolson@arizona.edu.
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  Data: <searchLink fieldCode="JN" term="%222985121R%22">The Journal of biological chemistry</searchLink> [J Biol Chem] 2025 Dec; Vol. 301 (12), pp. 110855. <i>Date of Electronic Publication: </i>2025 Oct 22.
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  Data: Journal Article; Research Support, N.I.H., Extramural
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Elsevier+Inc%2E+on+behalf+of+American+Society+for+Biochemistry+and+Molecular+Biology%22">Elsevier Inc. on behalf of American Society for Biochemistry and Molecular Biology </searchLink><i>Country of Publication: </i>United States <i>NLM ID: </i>2985121R <i>Publication Model: </i>Print-Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>1083-351X (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2200219258%22">00219258 </searchLink><i>NLM ISO Abbreviation: </i>J Biol Chem <i>Subsets: </i>MEDLINE
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        Value: 10.1016/j.jbc.2025.110855
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      – Code: eng
        Text: English
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      – TitleFull: MYBPC3 N-terminal missense mutations linked to hypertrophic cardiomyopathy strengthen actin binding and enhance residue mobility.
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              M: 12
              Text: 2025 Dec
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