RIPK1 regulates β-cell fate via actions on gene expression and kinase signaling in a mouse model of β-cell self-reactivity.
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| Title: | RIPK1 regulates β-cell fate via actions on gene expression and kinase signaling in a mouse model of β-cell self-reactivity. |
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| Authors: | Contreras CJ; Department of Medicine, Roudebush VA Medical Center & Indiana University School of Medicine, Indianapolis, IN, USA.; Lilly Diabetes Center of Excellence, Indiana Biosciences Research Institute, Indianapolis, IN, USA., Mukherjee N; Lilly Diabetes Center of Excellence, Indiana Biosciences Research Institute, Indianapolis, IN, USA.; Department of Biochemistry & Molecular Biology, Indiana University School of Medicine, Indianapolis, IN, USA., Harris-Kawano A; Department of Biochemistry & Molecular Biology, Indiana University School of Medicine, Indianapolis, IN, USA., Mather EG; Lilly Diabetes Center of Excellence, Indiana Biosciences Research Institute, Indianapolis, IN, USA., Amarsaikhan N; Lilly Diabetes Center of Excellence, Indiana Biosciences Research Institute, Indianapolis, IN, USA., Davis C; Department of Pediatrics, Indiana University School of Medicine, Indianapolis, IN, USA., Berryhill CA; Department of Pediatrics, Indiana University School of Medicine, Indianapolis, IN, USA., Peyton M; Department of Biostatistics & Health Data Science, Indiana University School of Medicine, Indianapolis, IN, USA., Kundu D; Lilly Diabetes Center of Excellence, Indiana Biosciences Research Institute, Indianapolis, IN, USA., Colglazier KA; Lilly Diabetes Center of Excellence, Indiana Biosciences Research Institute, Indianapolis, IN, USA., Miller AS; Lilly Diabetes Center of Excellence, Indiana Biosciences Research Institute, Indianapolis, IN, USA., Branco RCS; Lilly Diabetes Center of Excellence, Indiana Biosciences Research Institute, Indianapolis, IN, USA.; Department of Pediatrics, Indiana University School of Medicine, Indianapolis, IN, USA., Johnson TS; Lilly Diabetes Center of Excellence, Indiana Biosciences Research Institute, Indianapolis, IN, USA.; Department of Biostatistics & Health Data Science, Indiana University School of Medicine, Indianapolis, IN, USA., Angus SP; Department of Pediatrics, Indiana University School of Medicine, Indianapolis, IN, USA., You S; Lilly Diabetes Center of Excellence, Indiana Biosciences Research Institute, Indianapolis, IN, USA.; Université Paris Cité, Institut Cochin, CNRS, INSERM, Paris, France.; Center for Diabetes & Metabolic Diseases, Indiana University School of Medicine, Indianapolis, IN, USA., Cai EP; Lilly Diabetes Center of Excellence, Indiana Biosciences Research Institute, Indianapolis, IN, USA.; Center for Diabetes & Metabolic Diseases, Indiana University School of Medicine, Indianapolis, IN, USA., Templin AT; Department of Medicine, Roudebush VA Medical Center & Indiana University School of Medicine, Indianapolis, IN, USA. templin@iu.edu.; Lilly Diabetes Center of Excellence, Indiana Biosciences Research Institute, Indianapolis, IN, USA. templin@iu.edu.; Department of Biochemistry & Molecular Biology, Indiana University School of Medicine, Indianapolis, IN, USA. templin@iu.edu.; Center for Diabetes & Metabolic Diseases, Indiana University School of Medicine, Indianapolis, IN, USA. templin@iu.edu. |
| Source: | Cell death & disease [Cell Death Dis] 2026 Feb 12; Vol. 17 (1), pp. 220. Date of Electronic Publication: 2026 Feb 12. |
| Publication Type: | Journal Article |
| Journal Info: | Publisher: Nature Pub. Group Country of Publication: England NLM ID: 101524092 Publication Model: Electronic Cited Medium: Internet ISSN: 2041-4889 (Electronic) NLM ISO Abbreviation: Cell Death Dis Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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| ISSN: | 2041-4889 |
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| DOI: | 10.1038/s41419-026-08471-0 |
