IgA autoantibodies promote inflammation, Th17 polarization and fibrotic responses in hidradenitis suppurativa.

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Title: IgA autoantibodies promote inflammation, Th17 polarization and fibrotic responses in hidradenitis suppurativa.
Authors: Carmona-Rivera C; Systemic Autoimmunity Branch, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, MD, USA. carmelo.carmona-rivera@nih.gov., O'Neil LJ; Manitoba Centre for Proteomics and Systems Biology, Department of Internal Medicine, University of Manitoba, Winnipeg, MB, Canada., Patino-Martinez E; Systemic Autoimmunity Branch, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, MD, USA., Ambler WG; Systemic Autoimmunity Branch, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, MD, USA., Mallela T; Department of Dermatology, University of North Carolina, Chapel Hill, NC, USA., Hanata N; Systemic Autoimmunity Branch, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, MD, USA., Zadu AK; Department of Dermatology, Howard University College of Medicine, Washington, DC, USA., Jiang K; Biodata Mining and Discovery Section, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, MD, USA., Okoye GA; Department of Dermatology, Howard University College of Medicine, Washington, DC, USA.; Department of Dermatology, Johns Hopkins University School of Medicine, Baltimore, MD, USA., Byrd AS; Department of Dermatology, Howard University College of Medicine, Washington, DC, USA.; Department of Dermatology, Johns Hopkins University School of Medicine, Baltimore, MD, USA., Sayed CJ; Department of Dermatology, University of North Carolina, Chapel Hill, NC, USA., Kaplan MJ; Systemic Autoimmunity Branch, National Institute of Arthritis and Musculoskeletal and Skin Diseases, National Institutes of Health, Bethesda, MD, USA.
Source: Nature communications [Nat Commun] 2026 Mar 24; Vol. 17 (1). Date of Electronic Publication: 2026 Mar 24.
Publication Type: Journal Article
Journal Info: Publisher: Nature Pub. Group Country of Publication: England NLM ID: 101528555 Publication Model: Electronic Cited Medium: Internet ISSN: 2041-1723 (Electronic) Linking ISSN: 20411723 NLM ISO Abbreviation: Nat Commun Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:2041-1723
DOI:10.1038/s41467-026-70883-5