Shiga toxin increases intestinal transit to displace resident microbes and facilitate pathogen colonization.
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| Title: | Shiga toxin increases intestinal transit to displace resident microbes and facilitate pathogen colonization. |
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| Authors: | Odem MA; Department of Microbiology and Molecular Genetics, The University of Texas Health Science Center at Houston, Houston, Texas, United States of America., Simbassa SB; Department of Microbiology and Molecular Genetics, The University of Texas Health Science Center at Houston, Houston, Texas, United States of America.; Microbiology and Infectious Diseases Program, University of Texas MD Anderson Cancer Center UTHealth Houston Graduate School of Biomedical Sciences, Houston, Texas, United States of America., Alvarez CF; Department of Microbiology and Molecular Genetics, The University of Texas Health Science Center at Houston, Houston, Texas, United States of America.; Microbiology and Infectious Diseases Program, University of Texas MD Anderson Cancer Center UTHealth Houston Graduate School of Biomedical Sciences, Houston, Texas, United States of America., Jeilu O; Department of Microbiology and Molecular Genetics, The University of Texas Health Science Center at Houston, Houston, Texas, United States of America., Simar SR; Center for Infectious Diseases, The UTHealth Houston School of Public Health at Houston, Houston, Texas, United States of America., Bosserman R; Department of Microbiology and Molecular Genetics, The University of Texas Health Science Center at Houston, Houston, Texas, United States of America., Dutta S; Department of Microbiology and Molecular Genetics, The University of Texas Health Science Center at Houston, Houston, Texas, United States of America., Galdamez W; Department of Microbiology and Molecular Genetics, The University of Texas Health Science Center at Houston, Houston, Texas, United States of America.; Microbiology and Infectious Diseases Program, University of Texas MD Anderson Cancer Center UTHealth Houston Graduate School of Biomedical Sciences, Houston, Texas, United States of America., Ayele H; Center for Infectious Diseases, The UTHealth Houston School of Public Health at Houston, Houston, Texas, United States of America., Hanson BM; Center for Infectious Diseases, The UTHealth Houston School of Public Health at Houston, Houston, Texas, United States of America., Proctor DM; Department of Microbiology and Molecular Genetics, The University of Texas Health Science Center at Houston, Houston, Texas, United States of America.; Microbiology and Infectious Diseases Program, University of Texas MD Anderson Cancer Center UTHealth Houston Graduate School of Biomedical Sciences, Houston, Texas, United States of America., Krachler AM; Department of Microbiology and Molecular Genetics, The University of Texas Health Science Center at Houston, Houston, Texas, United States of America.; Microbiology and Infectious Diseases Program, University of Texas MD Anderson Cancer Center UTHealth Houston Graduate School of Biomedical Sciences, Houston, Texas, United States of America. |
| Source: | PLoS pathogens [PLoS Pathog] 2026 Mar 30; Vol. 22 (3), pp. e1014104. Date of Electronic Publication: 2026 Mar 30 (Print Publication: 2026). |
| Publication Type: | Journal Article |
| Journal Info: | Publisher: Public Library of Science Country of Publication: United States NLM ID: 101238921 Publication Model: eCollection Cited Medium: Internet ISSN: 1553-7374 (Electronic) Linking ISSN: 15537366 NLM ISO Abbreviation: PLoS Pathog Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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| ISSN: | 1553-7374 |
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| DOI: | 10.1371/journal.ppat.1014104 |
