Acute stress alleviates type 2 lung inflammation by restricting ILC2s through corticosterone-glucocorticoid receptor signaling.

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Title: Acute stress alleviates type 2 lung inflammation by restricting ILC2s through corticosterone-glucocorticoid receptor signaling.
Authors: Zhu L; Key Laboratory of Multi-Cell Systems, Center for Excellence in Molecular Cell Science, Shanghai Institute of Biochemistry and Cell Biology, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China., Huang Y; Key Laboratory of Multi-Cell Systems, Center for Excellence in Molecular Cell Science, Shanghai Institute of Biochemistry and Cell Biology, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China., Peng B; Department of Pulmonary and Critical Care Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China., Zhong S; Key Laboratory of Multi-Cell Systems, Center for Excellence in Molecular Cell Science, Shanghai Institute of Biochemistry and Cell Biology, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China., Zhang X; Department of Allergy, People's Hospital of Xinjiang Uygur Autonomous Region, Xinjiang Urumqi, China., Xiao F; Department of Allergy, People's Hospital of Xinjiang Uygur Autonomous Region, Xinjiang Urumqi, China., Asim D; Graduate school of Xinjiang Medical University, Urumqi, China., Pakezhati J; Graduate school of Xinjiang Medical University, Urumqi, China., Ma J; Graduate school of Xinjiang Medical University, Urumqi, China., Sun S; Department of Pulmonary and Critical Care Medicine, Ruijin Hospital, Shanghai Jiao Tong University School of Medicine, Shanghai, China., Xu J; School of Life Science, University of Science and Technology of China, Hefei, China., Cheng S; Key Laboratory of Multi-Cell Systems, Center for Excellence in Molecular Cell Science, Shanghai Institute of Biochemistry and Cell Biology, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China., Ma L; Key Laboratory of Multi-Cell Systems, Center for Excellence in Molecular Cell Science, Shanghai Institute of Biochemistry and Cell Biology, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China., Ling Z; Key Laboratory of Multi-Cell Systems, Center for Excellence in Molecular Cell Science, Shanghai Institute of Biochemistry and Cell Biology, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China., Yi C; Key Laboratory of Multi-Cell Systems, Center for Excellence in Molecular Cell Science, Shanghai Institute of Biochemistry and Cell Biology, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China., Lu D; Center for Pulmonary and Critical Care Medicine, Peoples Hospital of Xinjiang Uygur Autonomous Region, Xinjiang Urumqi, China. dongmeilu2009@sina.com., Liang J; Department of Allergy, People's Hospital of Xinjiang Uygur Autonomous Region, Xinjiang Urumqi, China. drliangjq@163.com., Sun B; Key Laboratory of Multi-Cell Systems, Center for Excellence in Molecular Cell Science, Shanghai Institute of Biochemistry and Cell Biology, Chinese Academy of Sciences, University of Chinese Academy of Sciences, Shanghai, China. bsun@sibs.ac.cn.; School of Life Science and Technology, ShanghaiTech University, Shanghai, China. bsun@sibs.ac.cn.
Source: Cellular & molecular immunology [Cell Mol Immunol] 2026 May; Vol. 23 (5), pp. 546-559. Date of Electronic Publication: 2026 Apr 02.
Publication Type: Journal Article
Journal Info: Publisher: Chinese Society of Immunology Country of Publication: China NLM ID: 101242872 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 2042-0226 (Electronic) Linking ISSN: 16727681 NLM ISO Abbreviation: Cell Mol Immunol Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:2042-0226
DOI:10.1038/s41423-026-01407-w