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Estrogen-induced epigenetic silencing of myotrophin reduces proliferation and is associated with chemosensitivity in hepatocellular carcinoma cells.

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Bibliographic Details
Title: Estrogen-induced epigenetic silencing of myotrophin reduces proliferation and is associated with chemosensitivity in hepatocellular carcinoma cells.
Authors: Muhammad JS; Research Institute of Medical and Health Sciences, University of Sharjah, Sharjah, United Arab Emirates.; College of Medicine, University of Sharjah, Sharjah, United Arab Emirates., Rah B; Research Institute of Medical and Health Sciences, University of Sharjah, Sharjah, United Arab Emirates., Khan N; Program in Biology, New York University Abu Dhabi, Abu Dhabi, United Arab Emirates., Janeeh A; Research Institute of Medical and Health Sciences, University of Sharjah, Sharjah, United Arab Emirates., Shafarin J; Research Institute of Medical and Health Sciences, University of Sharjah, Sharjah, United Arab Emirates., Talaat I; Research Institute of Medical and Health Sciences, University of Sharjah, Sharjah, United Arab Emirates.; College of Medicine, University of Sharjah, Sharjah, United Arab Emirates., Sadler KC; Program in Biology, New York University Abu Dhabi, Abu Dhabi, United Arab Emirates.; Center for Genomics and Systems Biology, New York University Abu Dhabi, Abu Dhabi, United Arab Emirates., Hamad M; Research Institute of Medical and Health Sciences, University of Sharjah, Sharjah, United Arab Emirates.; Department of Medical Laboratory Sciences, College of Health Sciences, University of Sharjah, Sharjah, United Arab Emirates.
Source: Epigenomics [Epigenomics] 2026 Apr; Vol. 18 (4), pp. 385-396. Date of Electronic Publication: 2026 Apr 08.
Publication Type: Journal Article
Journal Info: Publisher: Taylor & Francis Country of Publication: England NLM ID: 101519720 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1750-192X (Electronic) Linking ISSN: 1750192X NLM ISO Abbreviation: Epigenomics Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:1750-192X
DOI:10.1080/17501911.2026.2654575