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Hypoxia-induced BTN3A2 promotes glioma progression and chemoresistance via AKT/SP1/RAD51-mediated DNA damage.

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Title: Hypoxia-induced BTN3A2 promotes glioma progression and chemoresistance via AKT/SP1/RAD51-mediated DNA damage.
Authors: Xu Z; Department of Neurosurgery, The second Affiliated Hospital of Kunming Medical University, Kunming, China.; Department of Neurosurgery, Pu'er People's Hospital, Kunming University of Science and Technology Affiliated Hospital, Yunnan, China., Pu S; School of Biosciences, University of Birmingham, Birmingham, UK., Wu J; Department of Neurosurgery, Pu'er People's Hospital, Kunming University of Science and Technology Affiliated Hospital, Yunnan, China., Yuan S; Department of Respiratory and Critical Care Medicine, Pu'er People's Hospital, Kunming University of Science and Technology Affiliated Hospital, Yunnan, China., Huang X; Department of Neurosurgery, The second Affiliated Hospital of Kunming Medical University, Kunming, China., Tian J; Department of Neurosurgery, The second Affiliated Hospital of Kunming Medical University, Kunming, China., Li X; Department of Neurosurgery, Pu'er People's Hospital, Kunming University of Science and Technology Affiliated Hospital, Yunnan, China., Liu B; Department of Neurosurgery, The second Affiliated Hospital of Kunming Medical University, Kunming, China., Yu X; Department of Neurosurgery, The second Affiliated Hospital of Kunming Medical University, Kunming, China., Zhao J; Department of Neurosurgery, The second Affiliated Hospital of Kunming Medical University, Kunming, China., Shi F; Department of Neurosurgery, Pu'er People's Hospital, Kunming University of Science and Technology Affiliated Hospital, Yunnan, China. shifengcai@163.com., Chen X; Department of Neurosurgery, The second Affiliated Hospital of Kunming Medical University, Kunming, China. chenxi5@kmmu.edu.cn., Pu J; Department of Neurosurgery, The second Affiliated Hospital of Kunming Medical University, Kunming, China. pujun@kmmu.edu.cn.
Source: Cell death & disease [Cell Death Dis] 2026 Apr 11; Vol. 17 (1). Date of Electronic Publication: 2026 Apr 11.
Publication Type: Journal Article
Journal Info: Publisher: Nature Pub. Group Country of Publication: England NLM ID: 101524092 Publication Model: Electronic Cited Medium: Internet ISSN: 2041-4889 (Electronic) NLM ISO Abbreviation: Cell Death Dis Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Hypoxia-induced BTN3A2 promotes glioma progression and chemoresistance via AKT/SP1/RAD51-mediated DNA damage.
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  Data: <searchLink fieldCode="AU" term="%22Xu+Z%22">Xu Z</searchLink>; Department of Neurosurgery, The second Affiliated Hospital of Kunming Medical University, Kunming, China.; Department of Neurosurgery, Pu'er People's Hospital, Kunming University of Science and Technology Affiliated Hospital, Yunnan, China.<br /><searchLink fieldCode="AU" term="%22Pu+S%22">Pu S</searchLink>; School of Biosciences, University of Birmingham, Birmingham, UK.<br /><searchLink fieldCode="AU" term="%22Wu+J%22">Wu J</searchLink>; Department of Neurosurgery, Pu'er People's Hospital, Kunming University of Science and Technology Affiliated Hospital, Yunnan, China.<br /><searchLink fieldCode="AU" term="%22Yuan+S%22">Yuan S</searchLink>; Department of Respiratory and Critical Care Medicine, Pu'er People's Hospital, Kunming University of Science and Technology Affiliated Hospital, Yunnan, China.<br /><searchLink fieldCode="AU" term="%22Huang+X%22">Huang X</searchLink>; Department of Neurosurgery, The second Affiliated Hospital of Kunming Medical University, Kunming, China.<br /><searchLink fieldCode="AU" term="%22Tian+J%22">Tian J</searchLink>; Department of Neurosurgery, The second Affiliated Hospital of Kunming Medical University, Kunming, China.<br /><searchLink fieldCode="AU" term="%22Li+X%22">Li X</searchLink>; Department of Neurosurgery, Pu'er People's Hospital, Kunming University of Science and Technology Affiliated Hospital, Yunnan, China.<br /><searchLink fieldCode="AU" term="%22Liu+B%22">Liu B</searchLink>; Department of Neurosurgery, The second Affiliated Hospital of Kunming Medical University, Kunming, China.<br /><searchLink fieldCode="AU" term="%22Yu+X%22">Yu X</searchLink>; Department of Neurosurgery, The second Affiliated Hospital of Kunming Medical University, Kunming, China.<br /><searchLink fieldCode="AU" term="%22Zhao+J%22">Zhao J</searchLink>; Department of Neurosurgery, The second Affiliated Hospital of Kunming Medical University, Kunming, China.<br /><searchLink fieldCode="AU" term="%22Shi+F%22">Shi F</searchLink>; Department of Neurosurgery, Pu'er People's Hospital, Kunming University of Science and Technology Affiliated Hospital, Yunnan, China. shifengcai@163.com.<br /><searchLink fieldCode="AU" term="%22Chen+X%22">Chen X</searchLink>; Department of Neurosurgery, The second Affiliated Hospital of Kunming Medical University, Kunming, China. chenxi5@kmmu.edu.cn.<br /><searchLink fieldCode="AU" term="%22Pu+J%22">Pu J</searchLink>; Department of Neurosurgery, The second Affiliated Hospital of Kunming Medical University, Kunming, China. pujun@kmmu.edu.cn.
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  Data: <searchLink fieldCode="JN" term="%22101524092%22">Cell death & disease</searchLink> [Cell Death Dis] 2026 Apr 11; Vol. 17 (1). <i>Date of Electronic Publication: </i>2026 Apr 11.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Nature+Pub%2E+Group%22">Nature Pub. Group </searchLink><i>Country of Publication: </i>England <i>NLM ID: </i>101524092 <i>Publication Model: </i>Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>2041-4889 (Electronic) <i>NLM ISO Abbreviation: </i>Cell Death Dis <i>Subsets: </i>MEDLINE
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