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Glucagon-Like Peptide-1 Receptor Agonists Inhibit the Initiation of Toxic Amyloid-β42 Aggregation.

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Bibliographic Details
Title: Glucagon-Like Peptide-1 Receptor Agonists Inhibit the Initiation of Toxic Amyloid-β42 Aggregation.
Authors: Fallot LB; Department of Chemical and Biological Science and Engineering, United States Military Academy, West Point, New York 10996, United States of America.; Photonics Research Center, United States Military Academy, West Point, New York 10996, United States of America., Anderson CA; Department of Chemical and Biological Science and Engineering, United States Military Academy, West Point, New York 10996, United States of America.; Photonics Research Center, United States Military Academy, West Point, New York 10996, United States of America., Pinc JR; Department of Chemical and Biological Science and Engineering, United States Military Academy, West Point, New York 10996, United States of America.; Photonics Research Center, United States Military Academy, West Point, New York 10996, United States of America., Stevenson A; Department of Biology, Institute of Biochemistry, ETH Zurich, Otto Stern Weg 3, 8093 Zurich, Switzerland.; Bringing Materials to Life Initiative, ETH Zurich, Otto Stern Weg 3, 8093 Zurich, Switzerland., Schleck MC; Department of Chemical and Biological Science and Engineering, United States Military Academy, West Point, New York 10996, United States of America.; Photonics Research Center, United States Military Academy, West Point, New York 10996, United States of America., Hawryschuk E; Department of Chemical and Biological Science and Engineering, United States Military Academy, West Point, New York 10996, United States of America.; Photonics Research Center, United States Military Academy, West Point, New York 10996, United States of America., Li OZ; Department of Chemical and Biological Science and Engineering, United States Military Academy, West Point, New York 10996, United States of America.; Photonics Research Center, United States Military Academy, West Point, New York 10996, United States of America., Palchak JC; Department of Chemical and Biological Science and Engineering, United States Military Academy, West Point, New York 10996, United States of America.; Photonics Research Center, United States Military Academy, West Point, New York 10996, United States of America., Toole JR; Department of Chemical and Biological Science and Engineering, United States Military Academy, West Point, New York 10996, United States of America., Kubiak Ii RW; Department of Chemical and Biological Science and Engineering, United States Military Academy, West Point, New York 10996, United States of America., Dear AJ; Department of Biology, Institute of Biochemistry, ETH Zurich, Otto Stern Weg 3, 8093 Zurich, Switzerland.; Bringing Materials to Life Initiative, ETH Zurich, Otto Stern Weg 3, 8093 Zurich, Switzerland., Michaels TCT; Department of Biology, Institute of Biochemistry, ETH Zurich, Otto Stern Weg 3, 8093 Zurich, Switzerland.; Bringing Materials to Life Initiative, ETH Zurich, Otto Stern Weg 3, 8093 Zurich, Switzerland., Limbocker R; Department of Chemical and Biological Science and Engineering, United States Military Academy, West Point, New York 10996, United States of America.; Photonics Research Center, United States Military Academy, West Point, New York 10996, United States of America.
Source: Journal of the American Chemical Society [J Am Chem Soc] 2026 May 27; Vol. 148 (20), pp. 20582-20599. Date of Electronic Publication: 2026 May 14.
Publication Type: Journal Article
Journal Info: Publisher: American Chemical Society Country of Publication: United States NLM ID: 7503056 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1520-5126 (Electronic) Linking ISSN: 00027863 NLM ISO Abbreviation: J Am Chem Soc Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:1520-5126
DOI:10.1021/jacs.6c01289