Glucagon-Like Peptide-1 Receptor Agonists Inhibit the Initiation of Toxic Amyloid-β42 Aggregation.
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| Title: | Glucagon-Like Peptide-1 Receptor Agonists Inhibit the Initiation of Toxic Amyloid-β42 Aggregation. |
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| Authors: | Fallot LB; Department of Chemical and Biological Science and Engineering, United States Military Academy, West Point, New York 10996, United States of America.; Photonics Research Center, United States Military Academy, West Point, New York 10996, United States of America., Anderson CA; Department of Chemical and Biological Science and Engineering, United States Military Academy, West Point, New York 10996, United States of America.; Photonics Research Center, United States Military Academy, West Point, New York 10996, United States of America., Pinc JR; Department of Chemical and Biological Science and Engineering, United States Military Academy, West Point, New York 10996, United States of America.; Photonics Research Center, United States Military Academy, West Point, New York 10996, United States of America., Stevenson A; Department of Biology, Institute of Biochemistry, ETH Zurich, Otto Stern Weg 3, 8093 Zurich, Switzerland.; Bringing Materials to Life Initiative, ETH Zurich, Otto Stern Weg 3, 8093 Zurich, Switzerland., Schleck MC; Department of Chemical and Biological Science and Engineering, United States Military Academy, West Point, New York 10996, United States of America.; Photonics Research Center, United States Military Academy, West Point, New York 10996, United States of America., Hawryschuk E; Department of Chemical and Biological Science and Engineering, United States Military Academy, West Point, New York 10996, United States of America.; Photonics Research Center, United States Military Academy, West Point, New York 10996, United States of America., Li OZ; Department of Chemical and Biological Science and Engineering, United States Military Academy, West Point, New York 10996, United States of America.; Photonics Research Center, United States Military Academy, West Point, New York 10996, United States of America., Palchak JC; Department of Chemical and Biological Science and Engineering, United States Military Academy, West Point, New York 10996, United States of America.; Photonics Research Center, United States Military Academy, West Point, New York 10996, United States of America., Toole JR; Department of Chemical and Biological Science and Engineering, United States Military Academy, West Point, New York 10996, United States of America., Kubiak Ii RW; Department of Chemical and Biological Science and Engineering, United States Military Academy, West Point, New York 10996, United States of America., Dear AJ; Department of Biology, Institute of Biochemistry, ETH Zurich, Otto Stern Weg 3, 8093 Zurich, Switzerland.; Bringing Materials to Life Initiative, ETH Zurich, Otto Stern Weg 3, 8093 Zurich, Switzerland., Michaels TCT; Department of Biology, Institute of Biochemistry, ETH Zurich, Otto Stern Weg 3, 8093 Zurich, Switzerland.; Bringing Materials to Life Initiative, ETH Zurich, Otto Stern Weg 3, 8093 Zurich, Switzerland., Limbocker R; Department of Chemical and Biological Science and Engineering, United States Military Academy, West Point, New York 10996, United States of America.; Photonics Research Center, United States Military Academy, West Point, New York 10996, United States of America. |
| Source: | Journal of the American Chemical Society [J Am Chem Soc] 2026 May 27; Vol. 148 (20), pp. 20582-20599. Date of Electronic Publication: 2026 May 14. |
| Publication Type: | Journal Article |
| Journal Info: | Publisher: American Chemical Society Country of Publication: United States NLM ID: 7503056 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1520-5126 (Electronic) Linking ISSN: 00027863 NLM ISO Abbreviation: J Am Chem Soc Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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