Redundant and distinct mechanisms suppress innate immune activation during SARS-CoV-2 infection.
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| Title: | Redundant and distinct mechanisms suppress innate immune activation during SARS-CoV-2 infection. |
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| Authors: | Zhou F; Department of Pathology, University of Texas Medical Branch, Galveston, Texas, United States of America.; Galveston National Laboratory, Galveston, Texas, United States of America., Periasamy S; Department of Pathology, University of Texas Medical Branch, Galveston, Texas, United States of America.; Galveston National Laboratory, Galveston, Texas, United States of America., Jackson ND; Department of Disease Intervention and Prevention, Texas Biomedical Research Institute, San Antonio, Texas, United States of America., Cheng WS; Department of Neurology, Icahn School of Medicine at Mount Sinai, New York, New York, United States of America., Soto Acosta R; Department of Pathology, University of Texas Medical Branch, Galveston, Texas, United States of America.; Galveston National Laboratory, Galveston, Texas, United States of America., Tripathi A; Department of Pathology, University of Texas Medical Branch, Galveston, Texas, United States of America.; Galveston National Laboratory, Galveston, Texas, United States of America., Kedarinath K; Department of Pathology, University of Texas Medical Branch, Galveston, Texas, United States of America.; Galveston National Laboratory, Galveston, Texas, United States of America., Ilinykh PA; Department of Pathology, University of Texas Medical Branch, Galveston, Texas, United States of America.; Galveston National Laboratory, Galveston, Texas, United States of America., Ye C; Department of Disease Intervention and Prevention, Texas Biomedical Research Institute, San Antonio, Texas, United States of America., Chauhan S; Department of Pathology, University of Texas Medical Branch, Galveston, Texas, United States of America.; Galveston National Laboratory, Galveston, Texas, United States of America., Nudelman G; Department of Neurology, Icahn School of Medicine at Mount Sinai, New York, New York, United States of America., Zaslavsky E; Department of Neurology, Icahn School of Medicine at Mount Sinai, New York, New York, United States of America., Hao H; Department of Biochemistry and Molecular Biology, University of Texas Medical Branch, Galveston, Texas, United States of America., Widen SG; Department of Biochemistry and Molecular Biology, University of Texas Medical Branch, Galveston, Texas, United States of America., Martinez-Sobrido L; Department of Disease Intervention and Prevention, Texas Biomedical Research Institute, San Antonio, Texas, United States of America., Sealfon SC; Department of Neurology, Icahn School of Medicine at Mount Sinai, New York, New York, United States of America., Bukreyev A; Department of Pathology, University of Texas Medical Branch, Galveston, Texas, United States of America.; Galveston National Laboratory, Galveston, Texas, United States of America.; Department of Microbiology and Immunology, University of Texas Medical Branch, Galveston, Texas, United States of America.; Center for Biodefense and Emerging Viral Infections, University of Texas Medical Branch, Galveston, Texas, United States of America. |
| Source: | PLoS biology [PLoS Biol] 2026 May 20; Vol. 24 (5), pp. e3003808. Date of Electronic Publication: 2026 May 20 (Print Publication: 2026). |
| Publication Type: | Journal Article |
| Journal Info: | Publisher: Public Library of Science Country of Publication: United States NLM ID: 101183755 Publication Model: eCollection Cited Medium: Internet ISSN: 1545-7885 (Electronic) Linking ISSN: 15449173 NLM ISO Abbreviation: PLoS Biol Subsets: MEDLINE |
| Database: | MEDLINE Ultimate |
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