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Bilobalide increases SKCa current via 5-HT2BR-PLC-PKC pathway, causing hyperpolarization and enhanced EET production in human coronary artery endothelial cells.

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Title: Bilobalide increases SKCa current via 5-HT2BR-PLC-PKC pathway, causing hyperpolarization and enhanced EET production in human coronary artery endothelial cells.
Authors: Ruamyod K; Siriraj Vascular Physiology Research Center, Department of Physiology, Faculty of Medicine Siriraj Hospital, Mahidol University, 2 Wanglang Road, Bangkok, 10700, Thailand., Taokum P; Siriraj Vascular Physiology Research Center, Department of Physiology, Faculty of Medicine Siriraj Hospital, Mahidol University, 2 Wanglang Road, Bangkok, 10700, Thailand., Wongsatan N; Siriraj Vascular Physiology Research Center, Department of Physiology, Faculty of Medicine Siriraj Hospital, Mahidol University, 2 Wanglang Road, Bangkok, 10700, Thailand., Tapechum S; Department of Physiology, Faculty of Medicine Siriraj Hospital, Mahidol University, 2 Wanglang Road, Bangkok, 10700, Thailand., Watanapa WB; Siriraj Vascular Physiology Research Center, Department of Physiology, Faculty of Medicine Siriraj Hospital, Mahidol University, 2 Wanglang Road, Bangkok, 10700, Thailand. wattana.wat@mahidol.ac.th.
Source: BMC complementary medicine and therapies [BMC Complement Med Ther] 2026 May 28; Vol. 26 (1). Date of Electronic Publication: 2026 May 28.
Publication Type: Journal Article
Journal Info: Publisher: BioMed Central Country of Publication: England NLM ID: 101761232 Publication Model: Electronic Cited Medium: Internet ISSN: 2662-7671 (Electronic) Linking ISSN: 26627671 NLM ISO Abbreviation: BMC Complement Med Ther Subsets: MEDLINE
Database: MEDLINE Ultimate
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  Data: Bilobalide increases SK<subscript>Ca</subscript> current via 5-HT<subscript>2B</subscript>R-PLC-PKC pathway, causing hyperpolarization and enhanced EET production in human coronary artery endothelial cells.
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  Data: <searchLink fieldCode="AU" term="%22Ruamyod+K%22">Ruamyod K</searchLink>; Siriraj Vascular Physiology Research Center, Department of Physiology, Faculty of Medicine Siriraj Hospital, Mahidol University, 2 Wanglang Road, Bangkok, 10700, Thailand.<br /><searchLink fieldCode="AU" term="%22Taokum+P%22">Taokum P</searchLink>; Siriraj Vascular Physiology Research Center, Department of Physiology, Faculty of Medicine Siriraj Hospital, Mahidol University, 2 Wanglang Road, Bangkok, 10700, Thailand.<br /><searchLink fieldCode="AU" term="%22Wongsatan+N%22">Wongsatan N</searchLink>; Siriraj Vascular Physiology Research Center, Department of Physiology, Faculty of Medicine Siriraj Hospital, Mahidol University, 2 Wanglang Road, Bangkok, 10700, Thailand.<br /><searchLink fieldCode="AU" term="%22Tapechum+S%22">Tapechum S</searchLink>; Department of Physiology, Faculty of Medicine Siriraj Hospital, Mahidol University, 2 Wanglang Road, Bangkok, 10700, Thailand.<br /><searchLink fieldCode="AU" term="%22Watanapa+WB%22">Watanapa WB</searchLink>; Siriraj Vascular Physiology Research Center, Department of Physiology, Faculty of Medicine Siriraj Hospital, Mahidol University, 2 Wanglang Road, Bangkok, 10700, Thailand. wattana.wat@mahidol.ac.th.
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  Data: <searchLink fieldCode="JN" term="%22101761232%22">BMC complementary medicine and therapies</searchLink> [BMC Complement Med Ther] 2026 May 28; Vol. 26 (1). <i>Date of Electronic Publication: </i>2026 May 28.
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22BioMed+Central%22">BioMed Central </searchLink><i>Country of Publication: </i>England <i>NLM ID: </i>101761232 <i>Publication Model: </i>Electronic <i>Cited Medium: </i>Internet <i>ISSN: </i>2662-7671 (Electronic) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2226627671%22">26627671 </searchLink><i>NLM ISO Abbreviation: </i>BMC Complement Med Ther <i>Subsets: </i>MEDLINE
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        Value: 10.1186/s12906-026-05418-y
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      – Code: eng
        Text: English
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      – TitleFull: Bilobalide increases SKCa current via 5-HT2BR-PLC-PKC pathway, causing hyperpolarization and enhanced EET production in human coronary artery endothelial cells.
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            – D: 28
              M: 05
              Text: 2026 May 28
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