Editorial Expression of Concern: Recognition of the nonclassical MHC class I molecule H2-M3 by the receptor Ly49A regulates the licensing and activation of NK cells.

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Title: Editorial Expression of Concern: Recognition of the nonclassical MHC class I molecule H2-M3 by the receptor Ly49A regulates the licensing and activation of NK cells.
Authors: Andrews DM; Cancer Immunology Program, Trescowthick Laboratories, Peter MacCallum Cancer Centre, St. Andrews Place, East Melbourne, Australia. daniel.andrews@petermac.org.; Sir Peter MacCallum Department of Oncology, University of Melbourne, Parkville, Australia. daniel.andrews@petermac.org., Sullivan LC; Department of Microbiology and Immunology, University of Melbourne, Parkville, Australia., Baschuk N; Cancer Immunology Program, Trescowthick Laboratories, Peter MacCallum Cancer Centre, St. Andrews Place, East Melbourne, Australia.; Sir Peter MacCallum Department of Oncology, University of Melbourne, Parkville, Australia., Chan CJ; Cancer Immunology Program, Trescowthick Laboratories, Peter MacCallum Cancer Centre, St. Andrews Place, East Melbourne, Australia.; Department of Pathology and Immunology, Monash University, Prahran, Australia., Berry R; Department of Biochemistry and Molecular Biology, School of Biomedical Sciences, Monash University, Clayton, Australia., Cotterell CL; Cancer Immunology Program, Trescowthick Laboratories, Peter MacCallum Cancer Centre, St. Andrews Place, East Melbourne, Australia.; Sir Peter MacCallum Department of Oncology, University of Melbourne, Parkville, Australia., Lin J; Department of Microbiology and Immunology, University of Melbourne, Parkville, Australia., Halse H; Cancer Immunology Program, Trescowthick Laboratories, Peter MacCallum Cancer Centre, St. Andrews Place, East Melbourne, Australia.; Sir Peter MacCallum Department of Oncology, University of Melbourne, Parkville, Australia., Watt SV; Cancer Immunology Program, Trescowthick Laboratories, Peter MacCallum Cancer Centre, St. Andrews Place, East Melbourne, Australia.; Sir Peter MacCallum Department of Oncology, University of Melbourne, Parkville, Australia., Poursine-Laurent J; Rheumatology Division, Department of Medicine, Washington University School of Medicine, St. Louis, Missouri, USA., Wang CR; Department of Microbiology and Immunology, Northwestern University, Chicago, Illinois, USA., Scalzo AA; Centre for Ophthalmology and Vision Science, University of Western Australia, Crawley, Australia.; Centre for Experimental Immunology, Lions Eye Institute, Nedlands, Australia., Yokoyama WM; Rheumatology Division, Department of Medicine, Washington University School of Medicine, St. Louis, Missouri, USA.; Howard Hughes Medical Institute, Washington University School of Medicine, St. Louis, Missouri, USA., Rossjohn J; Department of Biochemistry and Molecular Biology, School of Biomedical Sciences, Monash University, Clayton, Australia., Brooks AG; Department of Microbiology and Immunology, University of Melbourne, Parkville, Australia., Smyth MJ; Cancer Immunology Program, Trescowthick Laboratories, Peter MacCallum Cancer Centre, St. Andrews Place, East Melbourne, Australia. mark.smyth@petermac.org.; Sir Peter MacCallum Department of Oncology, University of Melbourne, Parkville, Australia. mark.smyth@petermac.org.; Department of Pathology and Immunology, Monash University, Prahran, Australia. mark.smyth@petermac.org.
Source: Nature immunology [Nat Immunol] 2026 Jun 29. Date of Electronic Publication: 2026 Jun 29.
Publication Type: Expression of Concern
Journal Info: Publisher: Nature America Inc Country of Publication: United States NLM ID: 100941354 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1529-2916 (Electronic) Linking ISSN: 15292908 NLM ISO Abbreviation: Nat Immunol Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:1529-2916
DOI:10.1038/s41590-026-02597-1