Nicotine drives lung cancer progression by suppressing cuproptosis through the LIAS-DLAT axis and promoting M2 macrophage polarization.

Saved in:
Bibliographic Details
Title: Nicotine drives lung cancer progression by suppressing cuproptosis through the LIAS-DLAT axis and promoting M2 macrophage polarization.
Authors: Yang YF; Department of Medical Education and Research, Kaohsiung Veterans General Hospital, Kaohsiung, Taiwan., Lin MH; Division of Chest Medicine, Kaohsiung Veterans General Hospital, Kaohsiung, Taiwan., Lin YY; Department of Biomedical Imagine and Radiological Sciences, National Yang Ming Chiao Tung University, Taipei, Taiwan., Chang PM; Institute of Biopharmaceutical Sciences, National Yang Ming Chiao Tung University, Taipei, Taiwan; Department of Oncology, Taipei Veterans General Hospital, Taipei, Taiwan., Kim CH; Molecular and Cellular Glycobiology Unit, Department of Biological Sciences, SungKyunKwan University, Suwon, Gyunggi-Do, Republic of Korea., Chang YC; Department of Biomedical Imagine and Radiological Sciences, National Yang Ming Chiao Tung University, Taipei, Taiwan. Electronic address: yuchanchang@nycu.edu.tw., Tsai KW; Department of Research, Taipei Tzu Chi Hospital, Buddhist Tzu Chi Medical Foundation, New Taipei City, Taiwan. Electronic address: tch33225@tzuchi.com.tw.
Source: Free radical biology & medicine [Free Radic Biol Med] 2026 Jul 14; Vol. 255, pp. 81-94. Date of Electronic Publication: 2026 Jul 14.
Publication Type: Journal Article
Journal Info: Publisher: Elsevier Science Country of Publication: United States NLM ID: 8709159 Publication Model: Print-Electronic Cited Medium: Internet ISSN: 1873-4596 (Electronic) Linking ISSN: 08915849 NLM ISO Abbreviation: Free Radic Biol Med Subsets: MEDLINE
Database: MEDLINE Ultimate
Description
ISSN:1873-4596
DOI:10.1016/j.freeradbiomed.2026.07.026