APA (7th ed.) Citation

PH, v. d. M., MC, d. L., SR, R., RJ, G., CA, B., T, O., & CD, D. (1998). Discontinuation of treatment with IFN-beta leads to exacerbation of experimental autoimmune encephalomyelitis in Lewis rats. Rapid reversal of the antiproliferative activity of IFN-beta and excessive expansion of autoreactive T cells as disease promoting mechanisms. Journal of neuroimmunology, 84(1), 14. https://doi.org/10.1016/s0165-5728(97)00207-5

Chicago Style (17th ed.) Citation

PH, van der Meide, de Labie MC, Ruuls SR, Groenestein RJ, Botman CA, Olsson T, and Dijkstra CD. "Discontinuation of Treatment with IFN-beta Leads to Exacerbation of Experimental Autoimmune Encephalomyelitis in Lewis Rats. Rapid Reversal of the Antiproliferative Activity of IFN-beta and Excessive Expansion of Autoreactive T Cells as Disease Promoting Mechanisms." Journal of Neuroimmunology 84, no. 1 (1998): 14. https://doi.org/10.1016/s0165-5728(97)00207-5.

MLA (9th ed.) Citation

PH, van der Meide, et al. "Discontinuation of Treatment with IFN-beta Leads to Exacerbation of Experimental Autoimmune Encephalomyelitis in Lewis Rats. Rapid Reversal of the Antiproliferative Activity of IFN-beta and Excessive Expansion of Autoreactive T Cells as Disease Promoting Mechanisms." Journal of Neuroimmunology, vol. 84, no. 1, 1998, p. 14, https://doi.org/10.1016/s0165-5728(97)00207-5.