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Discontinuation of treatment with IFN-beta leads to exacerbation of experimental autoimmune encephalomyelitis in Lewis rats. Rapid reversal of the antiproliferative activity of IFN-beta and excessive expansion of autoreactive T cells as disease promoting mechanisms.

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Title: Discontinuation of treatment with IFN-beta leads to exacerbation of experimental autoimmune encephalomyelitis in Lewis rats. Rapid reversal of the antiproliferative activity of IFN-beta and excessive expansion of autoreactive T cells as disease promoting mechanisms.
Authors: van der Meide PH; Department of Immunobiology, Biomedical Primate Research Centre, Rijswijk, The Netherlands. meide@bprc.nl, de Labie MC, Ruuls SR, Groenestein RJ, Botman CA, Olsson T, Dijkstra CD
Source: Journal of neuroimmunology [J Neuroimmunol] 1998 Apr 01; Vol. 84 (1), pp. 14-23.
Publication Type: Journal Article; Research Support, Non-U.S. Gov't
Journal Info: Publisher: Elsevier/North-Holland Country of Publication: Netherlands NLM ID: 8109498 Publication Model: Print Cited Medium: Print ISSN: 0165-5728 (Print) Linking ISSN: 01655728 NLM ISO Abbreviation: J Neuroimmunol Subsets: MEDLINE
Database: MEDLINE Ultimate
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PubType: Academic Journal
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  Data: Discontinuation of treatment with IFN-beta leads to exacerbation of experimental autoimmune encephalomyelitis in Lewis rats. Rapid reversal of the antiproliferative activity of IFN-beta and excessive expansion of autoreactive T cells as disease promoting mechanisms.
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  Data: <searchLink fieldCode="AU" term="%22van+der+Meide+PH%22">van der Meide PH</searchLink>; Department of Immunobiology, Biomedical Primate Research Centre, Rijswijk, The Netherlands. meide@bprc.nl<br /><searchLink fieldCode="AU" term="%22de+Labie+MC%22">de Labie MC</searchLink><br /><searchLink fieldCode="AU" term="%22Ruuls+SR%22">Ruuls SR</searchLink><br /><searchLink fieldCode="AU" term="%22Groenestein+RJ%22">Groenestein RJ</searchLink><br /><searchLink fieldCode="AU" term="%22Botman+CA%22">Botman CA</searchLink><br /><searchLink fieldCode="AU" term="%22Olsson+T%22">Olsson T</searchLink><br /><searchLink fieldCode="AU" term="%22Dijkstra+CD%22">Dijkstra CD</searchLink>
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  Data: <searchLink fieldCode="JN" term="%228109498%22">Journal of neuroimmunology</searchLink> [J Neuroimmunol] 1998 Apr 01; Vol. 84 (1), pp. 14-23.
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  Data: Journal Article; Research Support, Non-U.S. Gov't
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  Data: <i>Publisher: </i><searchLink fieldCode="PB" term="%22Elsevier%2FNorth-Holland%22">Elsevier/North-Holland </searchLink><i>Country of Publication: </i>Netherlands <i>NLM ID: </i>8109498 <i>Publication Model: </i>Print <i>Cited Medium: </i>Print <i>ISSN: </i>0165-5728 (Print) <i>Linking ISSN: </i><searchLink fieldCode="IS" term="%2201655728%22">01655728 </searchLink><i>NLM ISO Abbreviation: </i>J Neuroimmunol <i>Subsets: </i>MEDLINE
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        Value: 10.1016/s0165-5728(97)00207-5
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      – TitleFull: Discontinuation of treatment with IFN-beta leads to exacerbation of experimental autoimmune encephalomyelitis in Lewis rats. Rapid reversal of the antiproliferative activity of IFN-beta and excessive expansion of autoreactive T cells as disease promoting mechanisms.
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              Text: 1998 Apr 01
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              Y: 1998
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